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Why It’s So Hard to Keep Weight Off

Did you know that your fat cells are supposed to communicate? But as we age, it gets harder and harder for our fat cells to send and receive messages — the messages that tell fat cells to open up and release stubborn fat from your body. That's why your body hoards more and more fat over the years, making diet and exercise far less effective at maintaining your weight.

The Power Of Omega-7

I’m sure you’ve heard of a certain oil called “Omega 3”. And how it’s one of the healthiest kinds of oil to take on a daily basis.

The typical American diet contains plenty of omega-6s, but is woefully deficient in omega-3s and monounsaturated fats. This has led to a consensus to eat more foods rich in omega-3s (fish and some nuts) and monounsaturated fats (from olive oil and some nuts), less saturated fats, and no hydrogenated fats.

Scientists at Harvard Medical School and the Cleveland Clinic have been investigating a unique fatty acid that has not yet caught on in the mainstream. : Omega-7

Omega-7 is like a distant cousin to Omega 3. But potentially with much more power.

This novel fatty acid called omega-7 can help break the cycle of erratic blood sugar unpredictable lipid levels, excess fat gain, as well as enhance insulin sensitivity.

Ultra Omega Burn™

The Omega-7 of Choice

Ultra Omega BurnTM contains Omega-7, the unique fatty acid that helps fat-cells communicate with each other.

Allowing the “bad fat” in your body to no longer be stored, but rather to be released and shed. This makes it easier for the cells to keep the lines of communication open.

They believe Omega 7’s ability to keep inflammation levels healthy is the key to allowing your fat cells to be able to receive the signals it was intended to.

When researchers put fat cells in a petri dish and added Omega-7... They were able to actually see and measure the fatty acids leaving the cells!

It looks like a fine, pure, edible oil. Only a very small amount was needed in the body to start seeing results on many levels like this:

  • Encourage fat cells to communicate to aid in weight loss with proper diet and exercise*
  • Keep blood sugar levels in balance*
  • Maintain and promote clean healthy arteries*
  • Helps to maintain cholesterol levels*
  • Maintains proper digestive function by strengthening and lining the digestive tract*
  • Maintain skin healthy, looking radiant, plump and keep fine lines at bay*

Get your own dose of Omega-7 through Ultra Omega BurnTM today.

Get Ultra Omega BurnTM Today
References:

[] Cruz MM, et al. Palmitoleic acid (16:1n7) increases oxygen consumption, fatty acid oxidation and ATP content in white adipocytes. Lipids Health Dis. 2018 Mar 20;17(1):55. https://www.ncbi.nlm.nih.gov/pubmed/29554895

[] Duckett SK, Volpi-Lagreca G, Alende M, Long NM. Palmitoleic acid reduces intramuscular lipid and restores insulin sensitivity in obese sheep. Diabetes Metab Syndr Obes. 2014;7:553-63. https://www.ncbi.nlm.nih.gov/pubmed/25429233

[] Bernstein AM, Roizen MF, Martinez L. Purified palmitoleic acid for thereduction of high-sensitivity C-reactive protein and serum lipids: double-blinded, randomized, placebo controlled study. J Clin Lipidol. 2014;8(6):612-7. https://www.ncbi.nlm.nih.gov/pubmed/25499944

[] Yang ZH, Miyahara H, Hatanaka A. Chronic administration of palmitoleic acid reduces insulin resistance and hepatic lipid accumulation in KK-Ay Mice with genetic type 2 diabetes. Lipids Health Dis. 2011;10:120. https://www.ncbi.nlm.nih.gov/pubmed/21774832

[] Bernstein AM, Roizen MF, Martinez L. Purified palmitoleic acid for thereduction of high-sensitivity C-reactive protein and serum lipids: double-blinded, randomized, placebo controlled study. J Clin Lipidol. 2014;8(6):612-7. https://www.ncbi.nlm.nih.gov/pubmed/25499944

[] Stefan N, Kantartzis K, Celebi N, et al. Circulating palmitoleate strongly and independently predicts insulin sensitivity in humans. Diabetes Care. 2010;33(2):405-7

[] Cruz MM, et al. Palmitoleic acid (16:1n7) increases oxygen consumption, fatty acid oxidation and ATP content in white adipocytes. Lipids Health Dis. 2018 Mar 20;17(1):55. https://www.ncbi.nlm.nih.gov/pubmed/29554895

[] Cruz MM, et al. Palmitoleic acid (16:1n7) increases oxygen consumption, fatty acid oxidation and ATP content in white adipocytes. Lipids Health Dis. 2018 Mar 20;17(1):55. https://www.ncbi.nlm.nih.gov/pubmed/29554895

[] Cruz MM, et al. Palmitoleic acid (16:1n7) increases oxygen consumption, fatty acid oxidation and ATP content in white adipocytes. Lipids Health Dis. 2018 Mar 20;17(1):55. https://www.ncbi.nlm.nih.gov/pubmed/29554895

[] Duckett SK, Volpi-Lagreca G, Alende M, Long NM. Palmitoleic acid reduces intramuscular lipid and restores insulin sensitivity in obese sheep. Diabetes Metab Syndr Obes. 2014;7:553-63. https://www.ncbi.nlm.nih.gov/pubmed/25429233

[] Yang ZH, Miyahara H, Hatanaka A. Chronic administration of palmitoleic acid reduces insulin resistance and hepatic lipid accumulation in KK-Ay Mice with genetic type 2 diabetes. Lipids Health Dis. 2011;10:120. https://www.ncbi.nlm.nih.gov/pubmed/21774832

[] Nunes EA, Rafacho A. Implications of Palmitoleic Acid (Palmitoleate) On Glucose Homeostasis, Insulin Resistance and Diabetes. Curr Drug Targets. 2017;18(6):619-628. https://www.ncbi.nlm.nih.gov/pubmed/26648072

[] Bolsoni-Lopes A, et al. Palmitoleic acid (n-7) increases white adipocyte lipolysis and lipase content in a PPARα-dependent manner. Am J Physiol Endocrinol Metab. 2013;305(9):E1093-102. https://www.ncbi.nlm.nih.gov/pubmed/24022867

[] de Souza CO, Vannice GK, Rosa Neto JC, Calder PC. Is Palmitoleic Acid a Plausible Nonpharmacological Strategy to Prevent or Control Chronic Metabolic and Inflammatory Disorders? Mol Nutr Food Res. 2018;62(1). https://www.ncbi.nlm.nih.gov/pubmed/28980402

[] Çimen I, et al. Prevention of atherosclerosis by bioactive palmitoleate through suppression of organelle stress and inflammasome activation. Sci Transl Med. 2016;8(358):358ra126 https://www.ncbi.nlm.nih.gov/pubmed/27683551

[] Yang ZH, Miyahara H, Hatanaka A. Chronic administration of palmitoleic acid reduces insulin resistance and hepatic lipid accumulation in KK-Ay Mice with genetic type 2 diabetes. Lipids Health Dis. 2011;10:120.

[] Bueno-Hernández N, et al. Effect of Cis-palmitoleic acid supplementation on inflammation and expression ofHNF4γ, HNF4α and IL6 in patients with ulcerative colitis. Minerva Gastroenterol Dietol. 2017;63(3):257-263. https://www.ncbi.nlm.nih.gov/pubmed/28185444

[] de Souza CO, et al. Palmitoleic Acid has Stronger Anti-Inflammatory Potential in Human Endothelial Cells Compared to Oleic and Palmitic Acids. Mol Nutr Food Res. 2018. doi: 10.1002/mnfr.201800322 https://www.ncbi.nlm.nih.gov/pubmed/30102465

[] Erkkola, R. & Yang, B. Sea buckthorn oils: Towards healthy mucous membranes. AGROFood Industry Hi-Tech , 2003;53(3). http://www.scicompdf.se/membrasin/yang_erk_2003.pdf

[] Song IB, et al. Effects of 7-MEGA(TM) 500 on Oxidative Stress, Inflammation, and Skin Regeneration in H(2)O(2)-Treated Skin Cells. Toxicol Res. 2018;34(2):103-110. https://www.ncbi.nlm.nih.gov/pubmed/29686772

[] Akamatsu H, et al. The inhibition of free radical generation by human neutrophils through the synergistic effects of metronidazole with palmitoleic acid: a possible mechanism of action of metronidazole in rosacea and acne. Arch Dermatol Res. 1990;282(7):449-54. https://www.ncbi.nlm.nih.gov/pubmed/2150301

[] Yamamoto Y, et al. Palmitoleic acid calcium salt: a lubricant and bactericidal powder from natural lipids. J Oleo Sci. 2015;64(3):283-8. https://www.ncbi.nlm.nih.gov/pubmed/25757432

[] Yoon WJ, et al. Effect of palmitoleic acid on melanogenic protein expression in murine b16 melanoma. J Oleo Sci. 2010;59(6):315-9. https://www.ncbi.nlm.nih.gov/pubmed/20484837

[] Yang B, et al. Effect of dietary supplementation with sea buckthorn (Hippophaë rhamnoides) seed and pulp oils on the fatty acid composition of skin glycerophospholipids of patients with atopic dermatitis. J Nutr Biochem. 2000 Jun;11(6):338-40. https://www.ncbi.nlm.nih.gov/pubmed/11002130